Search results for "Ketoglutaric Acids"

showing 4 items of 4 documents

Stable Oxidative Cytosine Modifications Accumulate in Cardiac Mesenchymal Cells From Type2 Diabetes Patients

2018

Rationale: Human cardiac mesenchymal cells (CMSCs) are a therapeutically relevant primary cell population. Diabetes mellitus compromises CMSC function as consequence of metabolic alterations and incorporation of stable epigenetic changes. Objective: To investigate the role of α-ketoglutarate (αKG) in the epimetabolic control of DNA demethylation in CMSCs. Methods and Results: Quantitative global analysis, methylated and hydroxymethylated DNA sequencing, and gene-specific GC methylation detection revealed an accumulation of 5-methylcytosine, 5-hydroxymethylcytosine, and 5-formylcytosine in the genomic DNA of human CMSCs isolated from diabetic donors. Whole heart genomic DNA analysis reveale…

Male0301 basic medicinePhysiologyPopulationheartBiologyMixed Function OxygenasesCytosineMice03 medical and health sciencesProto-Oncogene ProteinsfibroblastsHuman Umbilical Vein Endothelial CellsAnimalsHumansMyocytes CardiacEpigeneticsEnzyme InhibitorseducationCells CulturedEpigenomicsDemethylationeducation.field_of_studyDNA methylationDNA methylation; epigenomics; fibroblasts; heart; hyperglycemia; metabolism; physiology; cardiology and cardiovascular medicineMesenchymal Stem CellsSettore MED/13 - ENDOCRINOLOGIABase excision repairMolecular biologyThymine DNA GlycosylaseMice Inbred C57BLHEK293 Cells030104 developmental biologyDNA demethylationDiabetes Mellitus Type 2epigenomicsDNA methylationKetoglutaric AcidshyperglycemiaThymine-DNA glycosylaseCardiology and Cardiovascular MedicineOxidation-ReductionmetabolismCirculation Research
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Influence of therapeutic and toxic doses of neuroleptics and antidepressants on energy metabolism of the isolated perfused rat brain.

1973

The isolated perfused rat brain was used for a comparative study of the effects of promazine, imipramine, monodesmethyl promazine and desipramine on cerebral energy metabolism. After perfusion for 30 min or 1 h the brain levels of the following substrates and metabolites were estimated: P-creatine, creatine, ATP, ADP, AMP, glycogen, glucose, glucose-6-P, fructose diphosphate, dihydroxyacetone-P, pyruvate, lactate, α-ketoglutarate, and ammonia. Drug concentrations of 5·10−6 M and 10−5 M in the perfusion medium caused a significant decrease of glucose-6-P alone. When the drug concentration was raised to a toxic range (10−4 M), reflected in the EEG by the pattern of secondary discharges, an ac…

Malemedicine.medical_specialtyImipraminePhosphocreatineBiologyPharmacologyCreatineImipramineAcetonechemistry.chemical_compoundOrganophosphorus CompoundsAmmoniaInternal medicineDesipramineTriosesmedicineAnimalsGlycolysisPyruvatesPromazinePromazinePharmacologyGlycogenDose-Response Relationship DrugDesipramineFructosephosphatesGlucosephosphatesBrainFructoseElectroencephalographyGeneral MedicineRibonucleotidesCreatineAntidepressive AgentsRatsPerfusionEndocrinologyGlucoseTranquilizing AgentschemistryLactatesKetoglutaric AcidsEnergy MetabolismPerfusionGlycolysismedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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The 2-oxoglutarate binding site of prolyl 4-hydroxylase. Identification of distinct subsites and evidence for 2-oxoglutarate decarboxylation in a lig…

1984

The structure and function of the 2-oxoglutarate binding site of prolyl 4-hydroxylase was studied by assaying the inhibitory potential of 24 selected aliphatic or aromatic compounds. All except one of them inhibited the enzyme competitively with respect to 2-oxoglutarate and noncompetitively with respect to Fe2+, the Ki values ranging from 0.8 microM to over 15 mM. The Ki values for the two most effective inhibitors, pyridine 2,5-dicarboxylate and 2,4-dicarboxylate, were about 0.8 microM and 2 microM, these compounds being the most potent inhibitors of prolyl 4-hydroxylase with respect to 2-oxoglutarate known so far. Only one of the compounds tested, 2-oxoadipinate, was able to support hydr…

chemistry.chemical_classificationCoordination sphereBinding SitesDecarboxylationStereochemistryIronProcollagen-Proline DioxygenaseChick EmbryoLigand (biochemistry)LigandsBiochemistryBinding CompetitiveDecarboxylationHydroxylationchemistry.chemical_compoundEnzymechemistryOxidoreductaseMoietyAnimalsKetoglutaric AcidsFerrous CompoundsBinding siteProtein BindingEuropean journal of biochemistry
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L‐Aspartate as a high‐quality nitrogen source in Escherichia coli : Regulation of L‐aspartase by the nitrogen regulatory system and interaction of L‐…

2020

Escherichia coli uses the C4-dicarboxylate transporter DcuA for L-aspartate/fumarate antiport, which results in the exploitation of L-aspartate for fumarate respiration under anaerobic conditions and for nitrogen assimilation under aerobic and anaerobic conditions. L-Aspartate represents a high-quality nitrogen source for assimilation. Nitrogen assimilation from L-aspartate required DcuA, and aspartase AspA to release ammonia. Ammonia is able to provide by established pathways the complete set of intracellular precursors (ammonia, L-aspartate, L-glutamate, and L-glutamine) for synthesizing amino acids, nucleotides, and amino sugars. AspA was regulated by a central regulator of nitrogen meta…

endocrine system diseasesNitrogenGlutaminePII Nitrogen Regulatory ProteinsNitrogen assimilationDeaminationGlutamic AcidBiologymedicine.disease_causeAspartate Ammonia-LyaseMicrobiology03 medical and health sciencesBacterial ProteinsAmmoniaEscherichia colimedicineProtein Interaction Domains and MotifsNucleotideMolecular BiologyEscherichia coliNitrogen cycle030304 developmental biologyDicarboxylic Acid Transporterschemistry.chemical_classificationAspartic Acid0303 health sciences030306 microbiologyEscherichia coli ProteinsAssimilation (biology)Gene Expression Regulation BacterialAmino acidEnzymechemistryBiochemistryMutationKetoglutaric AcidsMetabolic Networks and PathwaysMolecular Microbiology
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